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  Poster Hall

Tues-65 - Diverse Role of Porins and blaCTX-M in Mediating Ertapenem Resistance Among Carbapenem Resistant Enterobacterales

Scientific Poster Session IV - Encore

Encore Presentations
  Tuesday, October 15, 2024
  08:30 AM–10:00 AM

Abstract

Among carbapenem-resistant Enterobacterales (CRE) are diverse mechanisms, including those that are resistant to meropenem but susceptible to ertapenem, adding further complexity to the clinical landscape. This study investigates the emergence of ertapenem-resistant, meropenem-susceptible (ErMs) Escherichia coli and Klebsiella pneumoniae CRE across five hospitals in San Antonio, Texas, USA, from 2012 to 2018. The majority of the CRE isolates were non-carbapenemase producers (NCP; 54%; 41/76); 56% of all NCP isolates had an ErMs phenotype. Among ErMs strains, E. coli comprised the majority (72%). ErMs strains carrying blaCTX-M had, on average, 9-fold higher copies of blaCTX-M than CP-ErMs strains as well as approximately 4-fold more copies than blaCTX-M-positive but ertapenem- and meropenem-susceptible (EsMs) strains (3.7 vs. 0.9, p < 0.001). Notably, carbapenem hydrolysis was observed to be mediated by strains harboring blaCTX-M with and without a carbapenemase(s). ErMs also carried more mobile genetic elements, particularly IS26 composite transposons, than EsMs (37 vs. 0.2, p < 0.0001). MGE- ISVsa5 was uniquely more abundant in ErMs than either EsMs or ErMr strains, with over 30 more average ISVsa5 counts than both phenotype groups (p < 0.0001). Immunoblot analysis demonstrated the absence of OmpC expression in NCP-ErMs E. coli, with 92% of strains lacking full contig coverage of ompC. Overall, our findings characterize both collaborative and independent efforts between blaCTX-M and OmpC in ErMs strains, indicating the need to reappraise the term “non-carbapenemase (NCP)”, particularly for strains highly expressing blaCTX-M. To improve outcomes for CRE-infected patients, future efforts should focus on mechanisms underlying the emerging ErMs subphenotype of CRE strains to develop technologies for its rapid detection and provide targeted therapeutic strategies.

Presenting Author

Cody Black PharmD, PhD
UT Health San Antonio

Authors

Samantha Aguilar PharmD
University Health

Sarah Bandy PharmD, PhD
The University of Texas at Austin

Raymond Benavides BA
The University of Texas at Austin

Steven Dallas PhD
UT Health San Antonio

Christopher Frei PharmD, MS, FCCP, BCPS
The University of Texas at Austin College of Pharmacy and University of Texas Health San Antonio Long School of Medicine

Gerard Gawrys PharmD
University Health

Jim Koeller MS
University of Texas at Austin

Grace Lee PharmD, PhD
The University of Texas at Austin College of Pharmacy and University of Texas Health Science Center School of Medicine

Alvaro Moreira MD
UT Health San Antonio

Kevin Quidilla PharmD
The University of Texas at Austin

Kelly R Reveles PharmD, PhD, BCPS
The University of Texas at Austin College of Pharmacy

Dan Smelter PharmD, PhD
The University of Texas at Austin

Wonhee So PharmD
Western University of Health Sciences

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